Leukemia 2012 VOL. 26 NO. 1

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Spotlight Review

使用CXCR4和VLA- 4抑制剂进行造血干细胞和祖细胞动员

Mobilization of hematopoietic stem and progenitor cells using inhibitors of CXCR4 and VLA-4

小分子RNA在急性白血病中的作用：从生物学个体到临床捐赠者

MicroRNAs in acute leukemia: from biological players to clinical contributors

干细胞归巢及动员新展望：有关生物活性脂质作为有效趋化因子和阳离子多肽作为易忽视的SDF-1浓度调节因子的综述

A novel perspective on stem cell homing and mobilization: review on bioactive lipids as potent chemoattractants and cationic peptides as underappreciated modulators of responsiveness to SDF-1 gradients

心血管疾病中的干细胞和祖细胞动员

Mobilization of stem and progenitor cells in cardiovascular diseases

血管微生态：正常和恶性造血干细胞之源

The vascular niche: home for normal and malignant hematopoietic stem cells

骨髓增生异常综合征的microRNA出现异常

Deregulation of microRNAs in myelodysplastic syndrome

Reviews

复发和难治性多发性骨髓瘤治疗策略：重点研究新药领域的药物测序和再治疗

Treatment strategies in relapsed and refractory multiple myeloma: a focus on drug sequencing and ‘’retreatment|[rsquo]| approaches in the era of novel agents

Original Articles

Notch可诱导人T-细胞受体γδ+沿着一种平行，高度增殖和富含CD4 + CD8 +双阳性途径向胸腺细胞分化

Notch induces human T-cell receptor |[gamma]||[delta]||[plus]| thymocytes to differentiate along a parallel, highly proliferative and bipotent CD4 CD8 double-positive pathway

IDH1与IDH2突变对骨髓增生异常综合征不同的预后影响：Mayo Clinic 277例患者研究

Differential prognostic effect of IDH1 versus IDH2 mutations in myelodysplastic syndromes: a Mayo Clinic Study of 277 patients

AML1反式激活的PIG7，可促进AML1 - ETO融合基因白血病细胞的凋亡和分化

PIG7, transactivated by AML1, promotes apoptosis and differentiation of leukemia cells with AML1-ETO fusion gene

AMP依赖性蛋白激酶/哺乳动物雷帕霉素靶蛋白复合物1对T细胞急性淋巴细胞白血病的治疗作用

AMP-dependent kinase/mammalian target of rapamycin complex 1 signaling in T-cell acute lymphoblastic leukemia: therapeutic implications

单核细胞和T细胞协同有利于正常和滤泡性淋巴瘤B细胞的生长：IL- 15和CD40L信号的作用

Monocytes and T cells cooperate to favor normal and follicular lymphoma B-cell growth: role of IL-15 and CD40L signaling

造血干细胞移植条件反射可激活补体级联调节并诱导骨髓蛋白水解环境：生物活性脂质和可溶性C5b- C9作为归巢因素的新作用

Conditioning for hematopoietic transplantation activates the complement cascade and induces a proteolytic environment in bone marrow: a novel role for bioactive lipids and soluble C5b-C9 as homing factors